Os Tibiale Externum Syndrome
Os Tibiale Externum
🦶 Introduction
Os Tibiale Externum Syndrome (OTES)—also known as accessory navicular syndrome—is a painful condition that arises when an accessory ossicle (the os tibiale externum) located medial to the navicular bone becomes symptomatic. While present congenitally in approximately 4–14% of the population, symptoms typically manifest later due to mechanical irritation or increased stress on the posterior tibialis tendon (PTT), often contributing to medial foot pain and sometimes to dysfunctional biomechanics like flatfoot or tendon insufficiency
✅ Structured Scientific Summary
1. Definition
An extra ossicle adjacent to the navicular bone (inside the arch), usually integrated into the distal PTT
Symptoms—medial arch pain, swelling, and PTT irritation—collectively define os tibiale externum syndrome
2. Cause
Congenital persistence of a secondary ossification center that fails to fuse by adolescence
Symptom onset triggers include: trauma (e.g., ankle sprains), overuse, ill‑fitting footwear, and flatfoot biomechanics placing increased strain on the PTT
Os classification types (Geist):
Type I: small sesamoid in PTT—rarely symptomatic.
Type II: triangular ossicle connected via cartilage—most symptomatic.
Type III: fused cornuate tuberosity—prominent and occasionally painful
3. Classification
Geist classification outlines three types—as above
Type II is most clinically significant: the cartilaginous synchondrosis allows motion and pain, and portions of PTT may insert on it
4. Signs & Symptoms
Medial arch pain, often dull or throbbing, exacerbated by: weight-bearing, running, footwear pressure
Palpable bony prominence with localized swelling an
Tenderness along PTT, possible weakened midfoot posture or PTT dysfunction
Associated flatfoot (pes planovalgus) is common, often contributing to symptom development
Onset typically in adolescence or adulthood after ossification or increased activity
5. Current Treatment Trends
A. Conservative Management (≈70–80% success)
Rest, NSAIDs, ice, and activity modification—initial strategy
Immobilisation: short leg cast or boot for 4–6 weeks during acute flares
Physical therapy:
Strengthening PTT and intrinsic foot muscles
Stretching calf and posterior tibialis
Proprioception and gait retraining
Orthotic management (arch support, shoe modifications)
Imaging-confirmed interventions: guided corticosteroid injections, shockwave therapy (supported by recent case success)
B. Surgical Intervention (~20–30% of cases)
Indications: persistent pain, failed conservative management, Type II/III prominence, or PTT dysfunction
Procedures: excision of ossicle ± repair of PTT, Kinder procedure (osteotomy + tendon realignment)
Post-op rehab: early ROM; progressive strengthening and proprioception; return to activity typically in 8–12 weeks.